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Diagnostic algorithms, monitoring, prognostication, and therapy in chronic myeloid leukemia (CML): a proposal of the Austrian CML platform.
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To target the tumor, array placement is based on the individual patient's magnetic resonance imaging results.
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Second-line treatments after first-line gefitinib therapy in advanced nonsmall cell lung cancer.
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Only 30-35% of patients with NSCLC present with sufficiently localized disease at diagnosis that curative surgical resection may be attempted (stages IA and IB, IIA and IIB, and IIIA).
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High-resolution genomic profiling of childhood ALL reveals novel recurrent genetic lesions affecting pathways involved in lymphocyte differentiation and cell cycle progression.
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Antiangiogenic therapy is very expensive and potentially toxic even in carefully selected patients; hence, a detailed discussion with patients about its modest benefits versus the risks and costs is important.
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Untreated VACV infectedcells and uninfected cells are shown as controls in the right panel.
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Incisional Biopsy: the removal of a portion of the affected tissue, for examination, using a knife.
In the study, glioblastoma patients who died in 2010 (after the FDA approved bevacizumab for this condition) survived significantly longer than those who died of the disease in 2008.
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Meier survival curves were drawn for progression-free survival and were compared by means of a log-rank test.
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T790M mutation in pretreatment tumor samples from 10 (38%) of 26 patients with nonsmall cell lung cancer.
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Outcomes of stereotactic ablative radiotherapy for central lung tumours: a systematic review.
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In this context, clinical criteria, such as young age, lack of smoking history, or a documented preceding adenocarcinoma, may be used to select patients for testing.
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C) Flow cytometry was used to analyze the annexin V-positive population of HT29 or C2BBel cells.
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Accordingly, the development of a new EGFR TKI without adverse events of interstitial lung diseases or the discovery of predictive factors of intestinal lung disease is expected.
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Acquired Resistance of Lung Adenocarcinomas to Gefitinib or Erlotinib Is Associated with a Second Mutation in the EGFR Kinase Domain.
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Disclaimer: The materials published on the Clinical Care Options Sites reflect the views of the reviewers or authors of the CCO material, not those of Clinical Care Options, LLC, the accredited provider, or the companies providing educational grants.
Transesophageal endoscopic ultrasound-guided fine-needle aspiration (EUS-FNA) and endobronchial ultrasound-guided transbronchial needle aspiration (EBUS-TBNA) biopsy: a combined approach in the evaluation of mediastinal lesions.
Cancer cells were seeded in 96-well plates and were treated with different doses of gefitinib, metformin or both for 72 hours.
But Owing to the paucity of current data on gefitinib between defferent races, the possibility of differences in the toxicity and efficacy of gefitinib for ethnicity cannot be excluded.
Though Gliadel wafers are used by some for initial treatment, they have shown only a modest increase in median survival over placebo (13.
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Although annual low-dose CT scans have shown benefit in current and former smokers, widespread screening is not yet recommended for asymptomatic patients.
Gefitinib and their combination in T24 and UMUC3, respectively.
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Furthermore, levels of beta-catenin phosphorylation and nuclear PKM2 were correlated with grades of glioma malignancy and prognosis.
The difference in survival benefit may be diluted because placebo patients crossed over to sunitinib upon disease progression, and most of these patients subsequently responded to sunitinib.
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Such an approach would be possible only in a multinational trial because of the rareness of this disease.
Nevertheless, tumor tissue that needed for mutation analysis is frequently unavailable.
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Effect of epidermal growth factor receptor tyrosine kinase domain mutations on the outcome of patients with non-small cell lung cancer treated with epidermal growth factor receptor tyrosine kinase inhibitors.
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Evaluation of biologic end points and pharmacokinetics in patients with metastatic breast cancer after treatment with erlotinib, an epidermal growth factor receptor tyrosine kinase inhibitor.
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Physicians should be reminded that interstitial lung disease may also occur after chemotherapy, although it was not reported in this trial.
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Immunotherapy has recently emerged as a new treatment option for certain lung cancers.
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Three Cases of Non-Small Cell Lung Cancer, Which Simultaneously Expressed Activating Epidermal Growth Factor Receptor (EGFR) Mutations and EGFR-Tyrosine Kinase Inhibitor (TKI) Resistance Mutations (T790M) Before Gefitinib Therapy.
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First-line gefitinib for patients with advanced non-small-cell lung cancer harboring epidermal growth factor receptor mutations without indication for chemotherapy.
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Randomized phase II study of gefitinib versus erlotinib in patients with advanced non-small cell lung cancer who failed previous chemotherapy.
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Butein (20 μM) treatment induces morphologic changes of bladder cancer cells BLS(M) from elongated morphology to rounded epithelial-like cells, accompanied by downregulation of vimentin, and gaining of E-cadherin compared to untreated control cells, indicating the reversal of mesenchymal-like phenotype.
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In EGFR-mutated patients with acquired resistance to either gefitinib or erlotinib continued EGFR inhibition might be clinically beneficial, akin to the continued blockage of ErbB2 (HER2) with trastuzumab in ErbB2-amplified breast cancers.
Rozen SM, Nahabedian MY, Manson PN: Management strategies for pyoderma gangrenosum: case studies and review of literature.
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One issue with delaying second-line therapy has been the "drop-off" of patients who never get second-line therapy presumably due to disease-related complications and erosion of their performance status (PS) precluding subsequent therapy.
Given the efficacy of EGFR TKI therapy in previously treated patients, many groups have explored the use of erlotinib and gefitinib in the firstline setting.
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As with gefitinib and erlotinib, HKI-272 binds EGFR within its ATP-binding pocket.
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In resistant Bxpc-3 cells, the mRNA levels of MDR proteins were significantly down-regulated by gefitinib (5 μM), especially in high-dose gemcitabine-induced cancer cells.
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We show for the first time that conventional cytostatics induce the EGF-activated tyrosine kinase pathway leading to the induction of ATP-binding cassette protein mediated multidrug resistance in HCC.
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First-line Indolent (FIT) study presented at the American Society of Hematology (ASH) Annual Meeting demonstrated the continued improvement in progression-free survival (PFS) following ibritumomab consolidation therapy for patients with follicular B-cell non-Hodgkin's lymphoma who achieved a response to first-line therapy over chemotherapy alone.
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